ACE inhibitors and ARBs can cause which electrolyte disturbance?

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Multiple Choice

ACE inhibitors and ARBs can cause which electrolyte disturbance?

Explanation:
Blocking angiotensin II with ACE inhibitors or ARBs reduces aldosterone secretion from the adrenal glands. Aldosterone normally promotes potassium excretion in the distal nephron, so less aldosterone means less potassium loss and more potassium retained, leading to hyperkalemia. This risk is higher in patients with kidney impairment or those taking other potassium-sparing meds. Hyponatremia and hypokalemia aren’t typical effects of this mechanism, and hypercalcemia isn’t commonly associated with ACE inhibitors or ARBs.

Blocking angiotensin II with ACE inhibitors or ARBs reduces aldosterone secretion from the adrenal glands. Aldosterone normally promotes potassium excretion in the distal nephron, so less aldosterone means less potassium loss and more potassium retained, leading to hyperkalemia. This risk is higher in patients with kidney impairment or those taking other potassium-sparing meds. Hyponatremia and hypokalemia aren’t typical effects of this mechanism, and hypercalcemia isn’t commonly associated with ACE inhibitors or ARBs.

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